Read online Carcinogenesis Abstracts, 1978, Vol. 16: A Monthly Publication Sponsored by the National Cancer Institute (Classic Reprint) - George P Studzinski | PDF
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Abstract carcinogenesis is a multistep process involving mutation and the subsequent selective clonal expansion of the mutated cell. Chemical and physical agents including those that induce reative oxygen species can induce and/or modulate this multistep process.
Volume 68, issue s1 supplement: abstracts from the 36th annual meeting of the histiocyte society virtual sessions: october 1, 2020 and november 5, 2020 january 2021.
Authors who are affiliated with any of the institutions that are part of projekt deal and publish open access in molecular carcinogenesis are not required to pay an apc as per the agreement starting july 1, 2019. For more information, here is a recent article on wiley and projekt deal.
This volume was made possible, in part, through cooperative agreement cr advisory groups (iarc, 1977, 1978, 1979, 1982, 1983, 1987, 1988, 1991; chemical abstracts service registry number, the latest primary name and the iupac.
Abstract citations volume content graphics metrics export citation nasa/ads.
Over a century has elapsed since the first demonstration that.
The use of chemical compounds benefits society in a number of ways. Pesticides, for instance, enable foodstuffs to be produced in sufficient.
J carcinog, official publication of carcinogenesis press,united states.
Vascular endothelial growth factor (vegf) is a well known factor that induces angiogenesis.
Abstract carcinogenesis may result from the action of any one or a combination of chemical, physical, biologic, and/or genetic insults to cells. The process of carcinogenesis may be divided into at least three stages: initiation, promotion, and progression.
Hamilton sr, hyland j, mcavinchey d, boitnott jk (1983) effect of dietary beer or ethanol on experimental colonic carcinogenesis. Gastroenterology 84: (part 2) 1180 (abstract) henefer ep (1966) ethanol, 30 per cent and hamster pouch carcinogenesis.
Carcinogenesis is a multistage process, involving interplay between the cancer cells and their surrounding stromal cells [22][23] [24].
13: a monthly publication of the national cancer institute the secretary of health, education and welfare has determined that the publication of this periodical is necessary in the transaction of the public busi ness required by law of this department.
A stochastic model of carcinogenesis incorporating certain observations from chemical and radiation dose-response data.
A monthly publication sponsored by the national cancer institute.
Carcinogenesis abstracts, volume 13, issue 4 full view - 1975. Carcinogenesis abstracts, volume 11, issue 10 full view - 1973.
National cancer institute; categoria: lingua straniera - inglese; lunghezza: 390 pagine; anno: 1963.
Full text (pdf format only) and abstracts: january 1939 - december 1996.
Volume 11 1978 volume 10 1977 volume 9 1976 volume 8 1975 volume 7 1974 volume 6 1973 volume 5 1972 volume 4 1971 volume 3 1970 volume 2 1969 volume 1 1967-1968.
17: a monthly publication sponsored by the national cancer institute nutritional factors in the etiology of coronary heart disease, maturity-onset diabetes, diverticular disease, and dental caries are discussed. The possibility that nutrition plays a role in the etiology of malignant large bowel.
Colon carcinogenesis is a promising target for dietary intervention since polyphenols such as tea catechins can reach concentrations that far exceed their concentrations in other organs of the body. For colon cancer, the cancer chemopreventive activity of green tea has been attributed mostly to its egcg component.
Gastric cancer is the second most common cancer worldwide and the second most common cause of cancer-related deaths. Despite complete resection of gastric cancer and lymph node dissection, as well.
Primary liver cancer, which consists predominantly of hepatocellular carcinoma (hcc), is the fifth most common cancer worldwide and the third most common cause of cancer mortality. Hcc has several interesting epidemiologic features including dynamic temporal trends; marked variations among geographic regions, racial and ethnic groups, and between men and women; and the presence of several well.
The h 2-receptor antagonist, ranitidine, is among the most widely used pharmaceuticals to treat gastroesophageal reflux disease and peptic ulcers. While previous studies have demonstrated that amines can form n-nitrosamines when exposed to nitrite at stomach-relevant ph, n-nitrosamine formation from ranitidine, an amine-based pharmaceutical, has not been demonstrated under these.
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Cell division is controlled by stimulatory and inhibitory systems. The origin of cancer is monoclonal, and in order that a normal cell switches its phenotype and becomes a neoplastic cell.
Department of obstetrics and gynæcology, general hospital, oldham ol1 2jh, united kingdom.
O 6-methylguanine in blood leucocyte dna: an association with the geographic prevalence of gastric cancer and with low levels of serum pepsinogen a, a marker of severe chronic atrophic gastritis.
Journal of the chemical society a: inorganic, physical, theoretical (1966-1971) journal of the chemical society (resumed) (1926-1965) proceedings of the chemical society (1957-1964).
52: 1162–1170, 1992), we found that administration of a water extract of green tea leaves as the sole source of drinking fluid inhibited ultraviolet b light (uvb)-induced carcinogenesis in skh-1 mice previously initiated with 7,12-dimethylbenz[a]anthracene (dmba).
Carcinogenesis may result from the action of any one or a combination of chemical, physical, biologic, and/or genetic insults to cells. The process of carcinogenesis may be divided into at least three stages: initiation, promotion, and progression.
Sep 2, 1993 for chemical exposures, the chemical abstracts services registry number, ( 1978).
Carcinogenesis is a peer-reviewed medical journal in the field of cancer biology. It was established in 1980 and is published monthly by oxford university press.
A course of intravenous injections of corynebacterium parvum starting 4 days before a single intramuscular injection of methylcholanthrene markedly delayed or prevented the development of fibro-sacromas in cba mice. Similar treatment starting 4 weeks after injection of the carcinogen did not significantly alter the incidence of tumours.
It has been well established that environmental and occupational exposure to heavy metal causes cancer in several organs. Although the exact mechanism of heavy metal carcinogenesis remains elusive, metal-generated reactive oxygen species (ros) are essential.
Epidermoid carcinoma of the lateral border of the tongue can be induced in a standard strain of hamsters, applying dmba in acetone after traumatizing the area with a root canal broach. Employing the same technique in an inbred strain of hamsters resulted in the more rapid development of carcinomas. The inbred strain should be a useful model for experimental carcinogenesis of tongue.
Over a century has elapsed since the first demonstration that exposure to chemicals in coal tar can cause cancer in animals. These observations provided an essential causal mechanistic link between environmental chemicals and increased risk of cancer in human populations. Mouse models of chemical carcinogenesis have since led to the concept of multistage tumor development through distinct.
This collection of abstracts is part of a continuing series providing information on recent doctoral dissertations. The 16 titles deal with the following topics: the effects of teacher-directed lessons on fourth and fifth grade students; self-concept and the comprehension of scientific reading; the readability of junior college textbooks; the effects of prereading strategies on comprehension.
Abstract this review discusses the relevance of oxidative damage to metal-induced toxicity and carcinogenesis. Presented are important facts and mechanistic concepts on the capacity of selected transition metals, mainly ni, but also cu, co, cr, and briefly several others, to generate active oxygen species and other reactive intermediates under.
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